An example of nutrigenetics visithomepage in crohn's disease Catarina sousa guerreiro bsc, ms1, paula ferreira bsc2, lourdes tavares md3, paula moura santos md3, manuela neves bsc, phd4, miguel brito bsc, phd2 and mar cravo md, phd51department ofDietetics, escola superiorDe tecnologiaDa saDe lisboa, unidadeDe nutri e metabolismoDo institutoDe medicina molecularDa universidadeDe lisboa, lisboa, portugal2escola superiorDe tecnologiaDa saDe lisboa, lisboa, portugal3serviDe gastrenterologiaDo hospitalDe santa maria, epe, lisboa, portugal4instituto superiorDe agronomia,DepartamentoDe matem lisboa, portugal5serviDe gastrenterologiaDo instituto portuguDe oncologiaDe lisboa francisco gentil, epe, lisboa, portugalcorrespondence:Catarina sousa guerreiro, cientificaDeDiet escola superiorDe tecnologiaDa saDe lisboa, av.D.Jo ii, lote 4.69.01 1990096 Lisboa, Portugal.Top of pageabstractobjectives:The aim of this work was to study the interaction between genetic polymorphisms(Singlenucleotide polymorphisms, snps) of pro and antiinflammatory cytokines and fat intake on the risk ofDeveloping Crohn'sDisease (Cd) or modifyingDisease activity.Methods:Seven snps in interleukin 1(Il1), tumor necrosis factor alpha(Tnf lymphotoxin alpha(Lt and il6 genes were analyzed in 116 controls and 99 patients with cd. The type of fat intake was evaluated, and the interaction between SNPs beats by dre online store uk andDietary fat in modulatingDisease activity was analyzed. Finally, low intake of n3 PUFA and high n6 PUFA ratio in patients with the TNF 857 polymorphism were associated with higherDisease activity (Or 95 ci or 95 ci respectively).
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